This article answers exactly what percentage of sleep should be deep sleep, what that means biologically, how it changes with age, and what raises or lowers it. Use the Sleep Quality Score to assess your deep sleep indicators, and the Sleep Debt Calculator to confirm your overall sleep quantity is supporting adequate deep sleep time.

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The Direct Answer

Deep sleep (N3 slow-wave sleep) should make up 13–23% of total sleep time in healthy adults.

For a person sleeping 8 hours, that is approximately 62–110 minutes of deep sleep per night.

The most commonly cited clinical benchmark is 20% of total sleep time as a practical target — equating to:

• 7 hours sleep → approximately 84 minutes deep sleep
• 8 hours sleep → approximately 96 minutes deep sleep
• 9 hours sleep → approximately 108 minutes deep sleep

These are targets, not thresholds. Individual variation is real. Age is the single largest determinant of how much deep sleep you actually achieve (see the age table below). The Sleep Quality Score helps identify whether your current sleep is producing adequate deep sleep based on its downstream functional indicators — since most people cannot directly measure their sleep stages without clinical equipment or a wearable device.

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What Percentage of Sleep Should Be Deep Sleep: Full Breakdown

What Is Deep Sleep (N3)?

Deep sleep — clinically called Stage N3, slow-wave sleep (SWS), or delta sleep — is the deepest and most physically restorative stage of the sleep cycle.

Key facts about deep sleep:

1. It is the hardest stage to wake from. People woken from N3 experience significant sleep inertia — grogginess, confusion, and impaired performance that can last 20–30 minutes.
2. It occurs primarily in the first half of the night. N3 is front-loaded — the longest and deepest episodes occur in the first two sleep cycles (roughly the first 3–4 hours of sleep).
3. It is defined by slow, high-amplitude brain waves. EEG recordings during N3 show synchronised delta waves (0.5–2 Hz) — large, slow oscillations that are distinct from all other sleep stages.
4. It is not the same as REM sleep. A common confusion. Deep sleep (N3) is NREM sleep; REM sleep is a separate stage associated with dreaming and emotional processing. Both are essential; they serve different functions.
5. Its percentage declines significantly with age. A 20-year-old may spend 20–25% of sleep in N3. A 70-year-old may spend only 5–10%.

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The Four Sleep Stages: Where Deep Sleep Fits

Understanding the percentage requires understanding the full architecture:

Stage	Type	% of total sleep	Primary function
N1	NREM (light)	5–10%	Sleep transition; very light; easily disturbed
N2	NREM (light–medium)	45–55%	Memory consolidation; sleep spindles; K-complexes
N3	NREM (deep)	13–23%	Physical restoration; glymphatic clearance; growth hormone; immune function
REM	REM	20–25%	Emotional processing; procedural memory; dreaming

What this means in a typical 8-hour night:

• N1: 25–48 minutes
• N2: 216–264 minutes (3.6–4.4 hours)
• N3: 62–110 minutes (1–1.8 hours) ← the focus of this article
• REM: 96–120 minutes (1.6–2 hours)

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What Deep Sleep Actually Does: 7 Functions

Deep sleep is not just "rest." It performs specific biological functions that no other sleep stage replicates:

1. Glymphatic brain cleaning. The brain's waste clearance system — the glymphatic network — operates primarily during N3. Slow-wave oscillations drive cerebrospinal fluid pulses that flush beta-amyloid and tau protein (Alzheimer's-associated proteins) from brain tissue. Fultz et al. (Science, 2019) directly visualised this: each slow oscillation produces a corresponding CSF pulse that clears metabolic waste. See our Glymphatic System article for the full mechanism.

2. Growth hormone secretion. The majority of daily growth hormone (GH) release occurs during the first N3 episode of the night. GH drives tissue repair, muscle protein synthesis, fat metabolism, and immune cell production. Without adequate N3, GH secretion is reduced — with implications for physical recovery, body composition, and immune function.

3. Memory consolidation (declarative). Facts, events, and episodic memories are consolidated from short-term hippocampal storage to long-term cortical storage during N3 sleep. Disrupting deep sleep impairs next-day memory for newly learned factual information.

4. Immune system restoration. Cytokine production, immune cell proliferation, and immunological memory formation (including vaccine response) are all N3-dependent processes. Short or fragmented N3 suppresses these functions.

5. Cardiovascular recovery. During N3, heart rate is at its lowest, blood pressure drops maximally (the nocturnal dip), and the parasympathetic nervous system dominates. This is the primary cardiovascular recovery window. Non-dippers — people who do not achieve adequate N3 — have 2.7 times the cardiovascular event risk of normal dippers (Fan et al., Hypertension, 2021).

6. Metabolic regulation. N3-specific growth hormone secretion and cortisol nadir support overnight insulin sensitivity restoration. Tasali et al. (PNAS, 2008) demonstrated that selectively suppressing N3 (without reducing total sleep time) reduced insulin sensitivity by 25% — establishing N3 as an independent metabolic variable, not just a component of sleep duration.

7. Cellular repair. DNA damage accumulated during waking cell division is repaired during sleep, with the highest repair rates during N3. Chronic N3 deficiency accelerates cellular ageing through this mechanism.

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Deep Sleep Percentage by Age: The Reference Table

Deep sleep percentage declines progressively across the lifespan — one of the most consistent findings in sleep architecture research. This is partly a normal ageing process and partly a consequence of accumulated sleep disruption, medications, and health conditions over decades.

Age group	Expected N3 % of sleep	Expected N3 duration (8 hrs)
Children (6–12)	25–35%	120–168 min
Teenagers (13–17)	20–28%	96–134 min
Young adults (18–25)	18–25%	86–120 min
Adults (26–40)	15–23%	72–110 min
Middle-aged adults (41–60)	10–18%	48–86 min
Older adults (61–75)	6–12%	29–58 min
Elderly (75+)	3–8%	14–38 min

Sources: Van Cauter et al. (JAMA, 2000); Ohayon et al. (Sleep Medicine Reviews, 2004) — the largest meta-analysis of normative sleep architecture across 5,000+ participants.

Key points from this table:

• The decline from young adulthood to middle age is steep — approximately half of N3 percentage is lost between age 25 and age 60 under typical conditions.
• The decline is not fully inevitable. It is accelerated by alcohol, benzodiazepines, untreated sleep apnea, and irregular sleep timing — all modifiable.
• A 65-year-old who avoids these suppressors and maintains good sleep habits retains meaningfully more N3 than age-matched peers who do not.
• Children have dramatically high N3 — this is developmentally essential for physical growth, neural pruning, and immune system development.

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What Reduces Deep Sleep Percentage: 10 Causes

The following factors reduce N3 percentage — each through a specific biological mechanism:

1. Alcohol before bed

Mechanism: Alcohol suppresses the amplitude of slow-wave oscillations, reducing the mechanical driving force of glymphatic CSF pulses. Effect appears at all doses. Magnitude: Even 1–2 units within 4 hours of bedtime measurably reduces N3 slow oscillation amplitude (Ebrahim et al., 2013).

2. Benzodiazepines and Z-drugs (zolpidem, zopiclone)

Mechanism: These medications produce electrical activity resembling NREM sleep on gross EEG but suppress the genuine slow oscillations that define restorative N3. You appear to sleep; glymphatic clearance does not occur at normal rates. Magnitude: Studies consistently show 25–40% reduction in genuine slow oscillation power compared to equivalent natural N3 sleep.

3. Obstructive sleep apnea (OSA)

Mechanism: Apneic arousals terminate N3 episodes before completion. Each arousal resets the sleep stage to N1 or N2, preventing sustained deep sleep. Magnitude: Moderate-to-severe OSA (AHI >15) reduces N3 by 30–50% compared to non-apnea matched controls. Use the Sleep Apnea Risk Screener to assess your risk.

4. Irregular sleep timing

Mechanism: N3 is partly circadian-timed — the SCN coordinates its deepest expression with the first half of the biological night. Irregular timing disrupts this coordination, reducing N3 amplitude and duration. Magnitude: Rotating shift workers show 20–35% less N3 than day workers sleeping equivalent hours.

5. Aging

Mechanism: Progressive reduction in the amplitude of slow oscillations — driven by loss of the cortical neuronal populations that generate them — reduces both N3 duration and its restorative efficacy. Magnitude: See age table above. Approximately 2% of N3 percentage lost per decade from age 30 onwards under average conditions.

6. Caffeine too close to bedtime

Mechanism: Caffeine blocks adenosine receptors. Adenosine accumulation during waking is a prerequisite for both sleep pressure and N3 depth — reduced adenosine signalling reduces N3 intensity. Magnitude: Drake et al. (2013) found caffeine taken 6 hours before bed significantly reduced slow-wave sleep. Use the Caffeine Cutoff Calculator for your personalised cutoff.

7. Chronic sleep restriction

Mechanism: Cumulative sleep debt reduces N3 amplitude and disrupts the normal homeostatic regulation of slow-wave activity. The body prioritises brief N3 episodes at the expense of sustained depth. Magnitude: Two weeks of 6-hour sleep reduces N3 slow-wave activity amplitude by approximately 30% compared to 8-hour sleep (Van Dongen et al., 2003).

8. Elevated bedroom temperature

Mechanism: Core body temperature must fall for N3 to initiate and sustain. A warm bedroom prevents this fall, reducing N3 duration and depth. Magnitude: Bedrooms above 75°F (24°C) significantly increase nocturnal wakefulness and reduce N3 (Okamoto-Mizuno and Mizuno, 2012). Optimal: 65–68°F (18–20°C).

9. Antidepressants (SSRIs, SNRIs, tricyclics)

Mechanism: These medications primarily affect REM sleep, but their effects on serotonin and norepinephrine pathways can indirectly alter NREM architecture including N3 in some individuals. Magnitude: Highly variable by medication and individual — some SSRIs modestly increase N3 while reducing REM; others reduce both. Discuss sleep architecture concerns with your prescriber.

10. Chronic psychological stress

Mechanism: Elevated evening cortisol — the primary stress hormone — directly suppresses the slow-wave oscillations that define N3. The HPA axis and N3 are neurochemically antagonistic. Magnitude: Adults with high chronic stress scores show N3 reductions of 15–25% compared to low-stress matched controls.

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What Increases Deep Sleep Percentage: 8 Evidence-Based Strategies

1. Exercise — especially aerobic, earlier in the day

Evidence level: Tier 1 — strongest available How it works: Aerobic exercise increases adenosine accumulation, which drives both sleep pressure and N3 depth. Morning or afternoon exercise produces the largest N3 increase. Specific finding: A 2014 meta-analysis by Kredlow et al. found that regular aerobic exercise significantly increased slow-wave sleep across 66 studies. Action: 30–45 minutes of moderate aerobic exercise (running, cycling, swimming) before 5 PM. See our Exercise Timing article for full protocol.

2. Consistent sleep schedule

Evidence level: Tier 1 How it works: Circadian regularity stabilises the timing and amplitude of slow-wave oscillations. Irregular wake times disrupt the circadian coordination of N3, reducing both its duration and depth. Action: Same wake time seven days per week. Use the Bedtime Calculator to set the corresponding bedtime. See our Sleep Hygiene Guide.

3. Cool bedroom temperature (65–68°F / 18–20°C)

Evidence level: Tier 1 How it works: Core temperature must drop for N3 to sustain. A cool room accelerates this drop and prevents the temperature rises that cause arousal from N3. Action: Set thermostat, use a fan, or switch to breathable natural-fibre bedding if AC is unavailable.

4. Eliminate alcohol before bed

Evidence level: Tier 1 How it works: Removing alcohol removes the primary suppressant of slow-wave oscillation amplitude. N3 rebounds toward normal within 1–3 alcohol-free nights. Action: Four-hour minimum buffer between last drink and bedtime. For N3 optimisation, alcohol-free sleep is significantly superior.

5. Treat obstructive sleep apnea with CPAP

Evidence level: Tier 1 How it works: CPAP eliminates apneic arousals that interrupt N3 episodes, allowing sustained deep sleep completion. N3 increases are among the most consistent benefits of CPAP treatment. Action: Use the Sleep Apnea Risk Screener first. If OSA is confirmed, CPAP adherence of >4 hours per night produces the largest N3 improvements.

6. Warm shower or bath 60–90 minutes before bed

Evidence level: Tier 2 How it works: Peripheral vasodilation from a warm bath redirects blood flow from the core to the skin, accelerating the core temperature drop that enables and sustains N3. Finding: Haghayegh et al. meta-analysis (2019) found this protocol reduced sleep onset latency by 10 minutes and improved overall sleep efficiency — effects likely partly mediated through improved N3 access. Action: 40–43°C (104–109°F) bath or shower, 60–90 minutes before target bedtime.

7. Magnesium glycinate (200–400 mg before bed)

Evidence level: Tier 2 How it works: Magnesium activates GABA receptors and antagonises NMDA receptors — both supporting the neurological conditions that allow slow-wave oscillation generation. Finding: Abbasi et al. (2012) found magnesium supplementation significantly improved sleep quality including slow-wave indicators in elderly insomniacs. Action: 200–400 mg magnesium glycinate (not oxide — lower bioavailability) 30–60 minutes before bed.

8. Reduce or eliminate benzodiazepines and Z-drugs (with prescriber guidance)

Evidence level: Tier 1 — mechanistically direct How it works: Discontinuing these medications allows genuine slow oscillation generation to resume. N3 rebound typically occurs within 1–2 weeks of discontinuation. Caution: Never discontinue these medications abruptly without prescriber guidance — withdrawal requires a structured taper. Discuss alternatives (CBT-I is the evidence-based first-line insomnia treatment that preserves N3 architecture).

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How to Know If You Are Getting Enough Deep Sleep

Most people cannot directly measure their N3 percentage without clinical polysomnography or a validated wearable. Here are the practical proxies:

Signs you are likely getting adequate deep sleep:

• You wake feeling physically restored — not just rested, but refreshed
• Morning muscle soreness from exercise is resolved within 24–36 hours
• You rarely wake during the night more than once
• You do not feel physical heaviness or fatigue by mid-afternoon
• Your immune function is normal — not getting sick significantly more than peers
• You maintain stable body weight without significant changes in appetite

Signs you may not be getting enough deep sleep:

• You sleep 7–8 hours but consistently wake feeling unrefreshed
• Physical recovery from exercise is slower than expected
• You gain weight despite stable diet and activity (metabolic N3 effects)
• You experience more frequent illness
• Morning grogginess lasts more than 30 minutes (sleep inertia)
• A wearable device consistently shows below 13% deep sleep

Use the Sleep Quality Score to get a structured assessment of these indicators. If multiple apply, use the Sleep Debt Calculator to confirm you are also getting adequate total sleep — insufficient duration reduces N3 time absolutely even when N3 percentage is maintained.

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Wearable Device Deep Sleep Accuracy: What to Know

Consumer wearables (Oura Ring, Apple Watch, Fitbit, Garmin) estimate sleep stages including deep sleep. Important caveats:

5 things to know about wearable deep sleep estimates:

1. Accuracy is moderate, not clinical. Studies comparing wearables to polysomnography find correlations of r = 0.50–0.75 for deep sleep staging — directionally useful, but not individually precise enough for clinical decision-making.

2. Wearables tend to underestimate deep sleep in younger adults and overestimate it in older adults. The algorithms are trained on population data and may not match your individual physiology.

3. Trends matter more than single nights. If your wearable consistently shows below 10% deep sleep over 30+ days, that is a meaningful signal worth investigating — even if any single night's figure is imprecise.

4. Wearable deep sleep does not equal PSG deep sleep. A reading of "45 minutes deep sleep" from a consumer device is not directly comparable to "45 minutes N3" from a sleep lab. Use wearable data to identify trends, not to diagnose.

5. Below 10% consistently over 14+ days warrants attention. Whether the true figure is 8% or 12% due to device error, a persistent reading below 10% is a signal to review the suppressors in the list above and consider the Sleep Apnea Risk Screener.

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Deep Sleep vs REM Sleep: Key Differences

A very common confusion. Here is the clearest comparison:

Feature	Deep sleep (N3)	REM sleep
Sleep type	NREM (Non-REM)	REM
Brain activity	Low frequency, high amplitude (delta waves)	High frequency, low amplitude (similar to waking)
Body state	Muscle tone reduced but present	Voluntary muscles paralysed (atonia)
Eye movement	None	Rapid, conjugate movements
When it occurs	First half of the night (cycles 1–2)	Second half of the night (cycles 3–5)
Primary function	Physical restoration, glymphatic clearance, immune repair	Emotional processing, procedural memory, creativity
% of total sleep	13–23%	20–25%
Dreaming	Rare (if present: short, thought-like)	Vivid, narrative, emotionally rich
Most suppressed by	Alcohol, benzodiazepines, OSA	Alcohol (second half), SSRIs, OSA (REM-predominant)

Both stages are essential. Focusing exclusively on deep sleep at the expense of REM — or vice versa — produces an incomplete picture. Short sleep disproportionately cuts REM (second-half of night); alcohol disproportionately cuts N3 first-half and REM second-half; OSA cuts both through fragmentation.

For the full REM picture, see our What Is REM Sleep article.

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Deep Sleep and Specific Health Outcomes: The Research Summary

Deep sleep and Alzheimer's disease risk:

• Every N3 slow oscillation produces a glymphatic CSF pulse that clears beta-amyloid
• One night of sleep deprivation increases brain beta-amyloid burden by ~5% (Shokri-Kojori et al., PNAS, 2018)
• The Lancet Commission on Dementia (2020) added sleep as a modifiable dementia risk factor — with N3 clearance function as the primary mechanistic basis
• Full article: Sleep and Dementia Risk

Deep sleep and type 2 diabetes risk:

• Selective N3 suppression (without total sleep reduction) reduces insulin sensitivity by 25% (Tasali et al., PNAS, 2008)
• This makes N3 an independent metabolic variable — not just a component of sleep duration
• Full article: Sleep Deprivation and Type 2 Diabetes

Deep sleep and blood pressure:

• Nocturnal blood pressure dipping occurs during N3 — the lowest sympathetic tone of any sleep stage
• Reduced N3 is independently associated with higher nocturnal blood pressure after adjusting for total sleep time (Fung et al., Hypertension, 2011)
• Full article: Sleep and Blood Pressure

Deep sleep and physical performance:

• Growth hormone secretion during N3 is the primary driver of overnight tissue repair and muscle protein synthesis
• Athletes sleeping 9–10 hours (extending N3 absolute time) show improved sprint speed, reaction time, and recovery rates (Mah et al., Sleep, 2011)

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Frequently Asked Questions

What percentage of sleep should be deep sleep?

Deep sleep (N3 slow-wave sleep) should account for 13–23% of total sleep time in healthy adults, with 20% as a practical clinical target. For an 8-hour sleep period, this equates to approximately 96 minutes of deep sleep. The percentage naturally declines with age — from 20–25% in young adults to 5–10% in adults over 65. Individual variation is normal within these ranges. Consistently below 13% — particularly if confirmed by wearable data over 30 or more days — warrants investigation into the specific suppressors listed in this article.

How much deep sleep do I need per night?

For most adults, 60–90 minutes of deep sleep per night is the functional target, depending on total sleep duration. Children need more — 120–168 minutes per night for optimal growth and development. Older adults typically achieve less — 29–58 minutes is common after age 60, though the biological requirement remains higher than what is routinely achieved. If you are sleeping 7–8 hours and consistently achieving less than 60 minutes of deep sleep (as estimated by a wearable), the suppressors most likely responsible are alcohol within 4 hours of bed, untreated sleep apnea, or significant chronic sleep restriction — all addressed in this article.

Why do I get so little deep sleep?

The most common causes of reduced deep sleep percentage are: alcohol before bed (suppresses slow-wave oscillation amplitude at all doses), obstructive sleep apnea (apneic arousals interrupt N3 episodes), benzodiazepines or Z-drugs (suppress genuine slow oscillations despite producing apparent NREM sleep), irregular sleep timing (disrupts circadian coordination of N3), chronic sleep restriction (reduces N3 amplitude through adenosine dysregulation), elevated bedroom temperature (prevents the core temperature drop needed for N3 sustenance), and aging (progressive reduction in the cortical populations generating slow oscillations). The Sleep Apnea Risk Screener addresses the most common undiagnosed cause.

Is deep sleep more important than REM sleep?

Both are essential — they perform different and non-interchangeable functions. Deep sleep (N3) primarily drives physical restoration: glymphatic brain cleaning, growth hormone secretion, immune repair, and cardiovascular recovery. REM sleep primarily drives psychological restoration: emotional memory processing, procedural skill consolidation, and creative associative thinking. Neither can substitute for the other. Short sleep disproportionately cuts REM (which is concentrated in the second half of the night). Alcohol disproportionately cuts N3. Complete sleep health requires adequate amounts of both.

Does waking up naturally mean you got enough deep sleep?

Waking naturally — without an alarm, feeling refreshed — is one of the strongest practical indicators that your sleep has completed its restorative stages, including N3. N3 is most concentrated in the first 3–4 hours of sleep, so if you complete your full natural sleep duration, you have almost certainly completed your major N3 episodes. Alarm-driven waking is more likely to interrupt the final REM-rich cycles of the night than N3 episodes, but it does reduce overall restorative quality. Waking naturally and feeling refreshed within 30 minutes — without caffeine — is a reliable positive indicator of adequate N3 completion.

How do I know if my deep sleep is too low?

The most reliable accessible method is a consistent wearable device reading below 13% of total sleep over 30 or more nights, combined with functional indicators: waking unrefreshed despite adequate hours, slow physical recovery from exercise, increased illness frequency, and persistent morning grogginess lasting more than 30 minutes. Clinical confirmation requires polysomnography. If you suspect low deep sleep, the Sleep Quality Score provides a structured functional assessment, and the Sleep Apnea Risk Screener screens for the most common treatable cause.

Can you get too much deep sleep?

Pathologically elevated N3 percentage is rare in otherwise healthy adults. High N3 is generally a sign of significant sleep debt being repaid — the brain rebounds with higher deep sleep intensity following deprivation. Chronically elevated deep sleep in the absence of prior deprivation (consistently above 30% of total sleep) warrants clinical evaluation, as it can occasionally be associated with specific neurological conditions. For the vast majority of people, "too much deep sleep" is not a practical concern — the overwhelming clinical problem is insufficient N3, not excess.

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The Bottom Line

Deep sleep should make up 13–23% of total sleep — approximately 60–110 minutes per 8-hour night.

It is the most physically restorative sleep stage, performing functions — glymphatic waste clearance, growth hormone secretion, immune repair, and cardiovascular recovery — that no other stage can replicate.

Its percentage declines with age, but that decline is accelerated by modifiable factors: alcohol before bed, untreated sleep apnea, benzodiazepines, irregular sleep timing, chronic sleep restriction, and elevated bedroom temperature.

Your 8-step action plan to protect and increase deep sleep:

1. Eliminate alcohol within 4 hours of bedtime — the single fastest-acting N3 improvement available
2. Screen for sleep apnea — use the Sleep Apnea Risk Screener
3. Keep your bedroom at 65–68°F (18–20°C) — core temperature drop is a prerequisite for N3
4. Fix your wake time — consistency is the circadian anchor for N3 architecture
5. Exercise aerobically in the morning or afternoon — adenosine accumulation deepens N3
6. Set your caffeine cutoff — use the Caffeine Cutoff Calculator
7. Assess your sleep quality indicators — use the Sleep Quality Score
8. Confirm your total duration is adequate — use the Sleep Debt Calculator — deep sleep percentage cannot compensate for insufficient total sleep time

The quality of your deep sleep is not a fixed biological trait. It is the output of a set of modifiable inputs — and every input on the list above is within your control tonight.

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Tools Referenced in This Article

• Sleep Quality Score — Assess the functional indicators of deep sleep adequacy without a sleep lab
• Sleep Debt Calculator — Confirm total sleep duration is supporting adequate absolute deep sleep time
• Sleep Apnea Risk Screener — Screen for the most common modifiable cause of reduced deep sleep
• Caffeine Cutoff Calculator — Set a personalised cutoff that protects adenosine-driven deep sleep depth
• Bedtime Calculator — Set a consistent bedtime to anchor circadian deep sleep coordination
• Sleep Hygiene Checklist — Audit all modifiable factors affecting deep sleep percentage
• Sleep Efficiency Calculator — Calculate sleep continuity metrics that reflect deep sleep fragmentation

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Related Reading

• What Is the Glymphatic System and Sleep — Health — The full biology of what deep sleep's slow oscillations actually do for the brain during N3
• What Is REM Sleep — Health — The companion stage to deep sleep — different functions, different suppressors, equally essential
• Sleep and Dementia Risk: What the Research Shows — Health — Why deep sleep's glymphatic clearance function is the primary biological basis for the sleep-Alzheimer's connection
• How Does Exercise Timing Affect Sleep Quality — Optimization — The evidence on using exercise to increase deep sleep percentage through adenosine and thermal mechanisms

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Disclaimer: This article is for educational and informational purposes only and does not constitute medical advice. The information provided is not a substitute for professional medical advice, diagnosis, or treatment. Always seek the guidance of a qualified healthcare provider with any questions you may have regarding a medical condition or sleep disorder. Never disregard professional medical advice or delay seeking it because of something you have read on this website.